Why did Louis XVI's older brother die in 1761

Question

Why did Louis XVI's older brother die in 1761

Answer 1

Louis' brother was 10 when he died.  It was not at all unusual for children to die in those times.  There were no antibiotics or vaccines for childhood diseases.  I haven't yet found a cause of death mentioned, but this most likely doesn't matter a whole lot.   Disease affected even the very rich.  

Answer 2

despite an extensive search I have been unable to find the cause of his death, probably though it was one of the many childhood deasiese that were around at that time, many of them were fatal, we don't have too much problem with them these days because of inoctulation programs around the world.  

Answer 3

The 10 year old was another brother of Louis XVI, and died incarcerated at 10 years old, from disease common to inmates.

The brother of Louis XVI was Louis XVIII, other name-Louis-Stanislas-Xavier was born on November 17, 1755 in the Palace of Versailles in France, the fourth son of Louis, Dauphin of France, and his wife, Marie-Josèphe of Saxony

Louis XVIII  died September 16, 1824) was a King of France and Navarre. The brother of Louis XVI, and uncle of Louis XVII, he ruled the kingdom from 1814 (although he dated his reign from the death of his nephew in 1795) until his own death in 1824, with a brief break in 1815 due to his flight from Napoleon during the Hundred Days.

Louis XVIII suffered from a severe case of gout,* which worsened with the years. At the end of his life, the King was wheelchair-bound most of the time.

Louis XVIII died on September 16, 1824, and was interred in the Saint Denis Basilica. His brother, the Comte d'Artois, succeeded him as Charles X.

*Gout (also called metabolic arthritis) is a disease created by a buildup of uric acid. In this condition, monosodium urate or uric acid crystals are deposited on the articular cartilage of joints, tendons and surrounding tissues due to elevated concentrations of uric acid in the blood stream. This provokes an inflammatory reaction of these tissues. Typically, persons with gout are obese, predisposed to diabetes and hypertension, and at higher risk of heart disease. Gout is more common in affluent societies due to a diet rich in proteins, fat, and alcohol. When it follows as a consequence of other health conditions such as renal failure, it is often regardless of the person's lifestyle.  Lin, et al have statistical evidence linking gout to lead poisoning, and lead level in the body is significantly correlated with urate excretion and gout.  It is known that lead sugar was used to sweeten wine, and that chronic lead poisoning is a cause of gout, which condition is then known as saturnine gout, because of its association with alcohol and excess.

Gout also can develop as co-morbidity of other diseases, including polycythaemia, leukaemia, intake of cytotoxics, obesity, diabetes, hypertension, renal disorders, and hemolytic anemia. This form of gout is often called secondary gout. Diuretics (particularly thiazide diuretics) have traditionally been blamed for precipitating attacks of gout because they compete at the same transporter, but a Dutch case-control study from 2006 appears to cast doubt on this conclusion.